Part of HP-06 — Neural Control & Coordination

Neural Control Disorders: NEET-Oriented Reference Table

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Neural and Sensory System Disorders

DisorderSystem AffectedPrimary CauseKey SymptomCorrection/Treatment
MyopiaEye (vision)Elongated eyeball OR excessive corneal/lens curvature → image in front of retinaCannot see distant objects clearlyConcave (diverging) lens
HypermetropiaEye (vision)Short eyeball OR flat cornea/lens → image behind retinaCannot see near objects clearlyConvex (converging) lens
PresbyopiaEye (lens)Age-related loss of lens elasticity → reduced accommodationCannot focus on near objects (affects people over 40)Bifocal lenses
AstigmatismEye (cornea)Irregular curvature of cornea (or lens) → different focal pointsBlurred/distorted vision at all distancesCylindrical lens
CataractEye (lens)Opacification (clouding) of lens — aging, UV, diabetesProgressive painless blurred visionSurgical lens replacement
GlaucomaEye (optic nerve)Elevated intraocular pressure → optic nerve damageProgressive peripheral vision loss → tunnel vision → blindnessEye drops (beta-blockers) or surgery
Night blindnessEye (rods)Vitamin A deficiency → rhodopsin insufficiencyCannot see in dim lightVitamin A supplementation
Conductive deafnessEar (ossicles)Otosclerosis (stapes fixation), perforated tympanic membraneHearing loss; bone conduction preservedSurgery, hearing aids
Sensorineural deafnessEar cochleaCNVIII\frac{cochlea}{CN VIII}Hair cell damage (noise, age), auditory nerve damageHearing loss; bone conduction also affectedCochlear implants
Parkinson's diseaseCNS (substantia nigra)Loss of dopaminergic neurons → dopamine deficitResting tremor, rigidity, bradykinesiaL-DOPA
Multiple sclerosisCNS (myelin)Autoimmune demyelination of CNS (oligodendrocytes)Variable: weakness, ataxia, optic neuritisImmunomodulatory drugs
Myasthenia gravisNMJAutoantibodies against nicotinic ACh receptorsFatigable muscle weakness, ptosis, diplopiaAChE inhibitors
Alzheimer's diseaseCNS (cortex, hippocampus)Amyloid plaques, neurofibrillary tangles; ACh system affectedMemory loss, cognitive declineSymptomatic (AChE inhibitors)
EpilepsyCNS (widespread)Neuronal hyperexcitability (GABA deficit or glutamate excess)Seizures (tonic-clonic, absence)Anti-epileptics (enhance GABA)
DepressionCNS (monoamine systems)Low serotonin (±norepinephrine, dopamine)Persistent low mood, anhedoniaSSRIs, SNRIs

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