Part of HP-01 — Digestion & Absorption

Bulleted Key Facts — Direct NEET Recall List

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Enzyme-Source-pH Facts:

  • Salivary amylase: salivary glands → starch → maltose → pH 6.8
  • Pepsin: chief cells (as pepsinogen) → proteins → large peptides → pH 1.5–2.0
  • Gastric lipase: chief cells → short-chain triglycerides → pH 4.0–5.0
  • Enterokinase: DUODENAL MUCOSA (not pancreas) → trypsinogen → trypsin → pH 7.0–8.0
  • Trypsin + Chymotrypsin: pancreas (as proenzymes) → proteins/peptides → smaller peptides → pH 7.5–8.5
  • Carboxypeptidase: pancreas (as procarboxypeptidase) → C-terminal amino acid cleavage → pH 7.5–8.0
  • Elastase: pancreas (as proelastase) → elastin → pH 8.0–8.5
  • Pancreatic lipase: pancreas (active) → emulsified triglycerides → FA + monoglycerides → pH 7.5–8.0
  • Pancreatic amylase: pancreas (active) → starch + glycogen → maltose → pH 7.0–8.0
  • Maltase/Sucrase/Lactase: brush border → disaccharides → monosaccharides → pH 7.0–8.0

Bile — Critical Distinction:

  • Produced: liver. Stored: gallbladder. Released by: CCK.
  • Contains: bile SALTS (not enzymes) → emulsification ONLY
  • Bile has ZERO enzymatic activity

Absorption Routes:

  • Glucose + amino acids → active transport (SGLT, Na-dependent) → blood capillaries → portal vein → liver
  • Fats (fatty acids + glycerol) → reassembled as triglycerides → chylomicrons → lacteals → thoracic duct → left subclavian vein (bypass liver)
  • Water → osmosis → large intestine → blood capillaries
  • Fat-soluble vitamins (A, D, E, K) → lacteals → thoracic duct (same as fats)

Structural Features:

  • Stomach: rugae (longitudinal mucosal folds → expansion + secretory surface)
  • Small intestine: villi + microvilli (brush border) → 600-fold surface area amplification
  • Large intestine: haustra (sacculations → slow transit for water absorption)

GI Hormones:

  • Gastrin: G-cells, pyloric stomach → stimulates HCl + pepsinogen
  • Secretin: S-cells, duodenum, trigger = acid → stimulates pancreatic bicarbonate
  • CCK: I-cells, duodenum, trigger = fats + amino acids → bile release + pancreatic enzymes
  • GIP: K-cells, duodenum, trigger = fats + glucose → inhibits gastric acid + stimulates insulin

Disorders:

  • Kwashiorkor: protein deficiency → oedema, distended belly (low albumin → low oncotic pressure → fluid leaks)
  • Marasmus: total calorie + protein deficiency → extreme wasting, NO oedema
  • Jaundice: excess bilirubin → yellow skin + sclera
  • Vomiting: medullary vomiting centre (medulla oblongata)

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