Part of HP-02 — Breathing & Exchange of Gases

Breathing and Gas Exchange — Respiratory Disorders Summary

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Respiratory Disorders at a Glance

DisorderAetiology (Cause)Core PathologyHallmark SymptomsLung Volume ChangesReversible?
AsthmaAllergens, exercise, infection → airway inflammation, bronchospasmReversible narrowing of bronchi/bronchioles; mucus hypersecretionWheezing, breathlessness, expiratory difficulty↑RV (air trapping), ↓FEV1Yes (bronchodilators)
EmphysemaChronic smoking (most common) → protease-mediated alveolar wall destructionIrreversible loss of alveolar walls and elastic tissueProgressive dyspnoea, barrel chest, reduced exercise tolerance↑RV, ↑TLC, ↑FRC, ↓VC, ↓FEV1No
SilicosisChronic inhalation of silica (SiO2) dust — mining, stone cutting, sandblastingMacrophage-mediated lung fibrosis; thickened alveolar wallsProgressive dyspnoea, reduced lung compliance, cough↓TLC, ↓VC (restrictive)No
AsbestosisLong-term inhalation of asbestos fibres — construction, shipbuildingLung fibrosis + pleural plaquesProgressive dyspnoea, dry cough↓TLC, ↓VC (restrictive)No

Key Distinctions for NEET

  • Asthma vs Emphysema: Asthma is reversible (airway narrowing); emphysema is irreversible (structural alveolar destruction). Barrel chest is emphysema, not asthma.

  • Silicosis vs Asbestosis: Both cause fibrosis. Asbestosis additionally carries a high risk of mesothelioma (pleural cancer) and lung cancer. Different causative agents: silica vs asbestos fibres.

  • Obstructive vs Restrictive pattern: Asthma and emphysema = obstructive (↑RV, ↓FEV1/FVC). Fibrotic diseases = restrictive (↓TLC, ↓VC, preserved FEV1/FVC ratio).

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